Allergy drugs will cure a diabetes and adiposity.

It is not so often possible to meet an immunology mentioned along with type 2 diabetes.
However scientists from Harvard Medical School researchers have gone on this way, applying a medicine against an allergy to adiposity and diabetes treatment. The medications Zaditor and ?romolyn stabilize a population of inflammatory immune cells called mast cells.

Other research has shown that regulatory T cell; provide communication between metabolic and immune systems, being responsible for inflammatory processes in fat tissue. The fat tissue at mice with stability to insulin differs decrease in level of these cells, in comparison with high quantity of macrophages (they develop the inflammatory substances burning hostile cells).
At type 1 diabetes immune cells attack pancreas cells, reducing insulin generation. At type 2 diabetes body cells, as a result of metabolism infringement, become deaf to the signals of insulin, stimulating consumption of glucose.
Mast cells facilitate healing of the damaged tissues, allowing improving of the blood-groove. However their surplus leads to leak molecular "garbage" into the tissue, causing inflammatory processes. Still earlier scientific have found out that the fat tissue of mice with a diabetes contains surplus of these cells.
For hypothesis check they began to give to mice allergy drugs, having divided rodents into four groups: control, group of a healthy food and group of two preparations, in one of which mice also was limited in a food. The third group has considerably dumped weight and has improved indicators of stability to insulin. In the fourth group the absolute recovery was observed. Then scientists have bred mice, whose bodies do not generate mast cells, and it has appeared that they were steady against a diabetes and adiposity.
Harvard scientists have defined that the lack regulatory T cells in fat tissue of mice with diabetes is the seed of disease. Their quantity was in inverse proportion to quantity of macrophages. Now they assume that the inflammation caused by macrophages, results of resistance to insulin. Regulatory T cells, in turn, serve as an inflammation regulator with normal weight of a body.